• The Molecule of the Month home page - MOTM Bristol
  • Molecule of the Month | Media Citizen's Blog
  • PDB Molecule of the Month - Wilson Laboratory

The Molecule of the Month home page, based at the School of Chemistry, University of Bristol

Molecule of the Month by John Emsley

Molecule of the Month | Knippium Chemistry

Posts about Molecule of the Month written by ecknipp
"Acids don't work like which repair skin by kick-starting inflammation," says an assistant clinical professor of dermatology at Boston University School of Medicine. "They dissolve the upper layer of cells to trigger repair." Plus, unlike with scrubs, the skin is not damaged by abrasives. So if you're sensitive, acids are actually a wise choice. Look for formulas that combine them with an , which slows their penetration into skin. (We like ) And don't rush in. "Irritation can take a while to show up," says Hirsch. "You may get to day four, then all of a sudden your skin reacts." Take a month to ramp up to daily use.

Molecule of the Month: N2S2 - University of Bristol

If you wish to contribute a Molecule of the Month page, just the URL and I'll add you to the list at the next opportunity. (There's generally a 2-3 month waiting list, so this gives you plenty of time to write and polish your page).


Molecule of the Month Digitalis

Molecule of the Month on CRISPR/Cas9 - …
From an initial glance at these results, one might conclude that there is a direct S-S bond. However further examination of orbitals 1 and 2, ( in a plane perpendicular to the molecular plane and passing through both S atoms), shows that this is not so, as can be seen in Figure 2.

The Molecule of the Month by scientist, author and artist Dr
Paul May and Simon Cotton have compiled a set of some of the most important and interesting molecules from this website, as well as many more that have never been published before, and published them as a book that you can buy from the or from other online retailers.

Molecule of the Month – March 2015 | Maxim's …

Antibodies are our molecular watchdogs, waiting and watching for viruses, bacteria and other unwelcome visitors. Antibodies circulate in the blood, scrutinizing every object that they touch. When they find an unfamiliar, foreign object, they bind tightly to its surface. In the case of viruses, like rhinovirus or poliovirus presented last month in the Molecule of the Month, a coating of bound antibodies may be enough to block infection. Antibodies alone, however, are no match for bacteria. When antibodies bind to a bacterial surface, they act as markers alerting the other powerful defensive mechanisms available in the immune system.

Wikipedia:WikiProject Biophysics/Molecule of the Month

Above is a drop-down alphabetical list of all the molecules. Click on your molecule of choice to take you directly to it in the chronological table below.

The Molecule of the Month home page

T-Cell Receptor
Viruses are one of the major dangers that we face in everyday life, so our immune system has powerful methods to fight them. Our cells call for help when they become infected, by displaying little pieces of the viruses on their surface. When the immune system finds these viral peptides, it quickly kills the infected cell and the viruses inside. Last month, we saw how an infected cell displays viral peptides using . This month, we will look at the T-cell receptor, the protein that recognizes these viral peptides.

eLiquid Manufacturing - Molecule Labs

HIV Envelope Glycoprotein
Viruses are faced with a tricky problem: they need to get inside cells, but cells are surrounded by a protective membrane. Enveloped viruses like HIV and influenza, which are themselves surrounded by a similar membrane, solve this problem by fusing with the cell membrane. The envelope glycoprotein (Env) of HIV performs the many complex steps needed for membrane fusion. First, it attaches itself to proteins on the surface of the cell. Then, it acts like a spring-loaded mousetrap and snaps into a new conformation that drags the virus and cell close enough that the membranes fuse. Finally, the HIV genome is released into the cell, where it quickly gets to work building new viruses.